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Directions

1-70 Exit 328 at Hwy 99 turn
Right at the first stop light.

At Hwys 24 & 99 intersection go
South to stop light, turn left.

Contact Info

Mailing address:
P.O. Box 84
Wamego, KS 66547

785-456-2040
wamegomuseum@wamego.net

Membership

Membership information is available here. Send the completed form along with membership amount to the address on the form.

Helpful Links

While visiting the Wamego area you might find these links helpful:

Columbian Theatre
Oz Museum
Pottawatomie Economic Development Corporation
Visit Wamego

 

 

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´╗┐Enteropathogenic Escherichia coli (EPEC) alters many functions of the host intestinal epithelia. Inflammation is initiated by activation of nuclear factor (NF)-kappaB, and paracellular permeability is enhanced via a Ca2+- and myosin light-chain kinase (MLCK)-dependent pathway. The aims of this study were to identify signaling pathways by Nike Blazer which EPEC triggers inflammation and to determine whether these pathways parallel or diverge from those that alter permeability. EPEC-induced phosphorylation and degradation of the primary inhibitor of NF-kappaB (IkappaBalpha) were tumor necrosis factor (TNF)-alpha and interleukin (IL)-1beta independent. In contrast to Salmonella typhimurium, EPEC-stimulated IkappaBalpha degradation and IL-8 expression did not require Ca2+. Instead, extracellular signal-regulated kinase (ERK)-1/2 was significantly and rapidly activated. ERK1/2 inhibitors attenuated IkappaBalpha degradation and IL-8 expression. Although ERK1/2 can activate MLCK, its inhibition had no impact on EPEC disruption of the tight junction barrier. In conclusion, Nike Outlet Milton EPEC-induced inflammation 1) is TNF-alpha and IL-1beta receptor independent, 2) utilizes pathways differently from S. typhimurium, 3) requires ERK1/2, and 4) employs signals that are distinct from those that alter permeability. This is the first time that EPEC-activated signaling cascades have been linked to independent functional consequences.  

 

 

 

 


 
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